021-51111890
购物车（）
sales@molnova.cn
LYN-1604 dihydrochloride
CAS No. 2310109-38-5
LYN-1604 dihydrochloride ( LYN-1604 2HCl（2216753-86-3 free basel); )
产品货号. M28580 CAS No. 2310109-38-5
LYN-1604 is a novel ULK1 activator.It inducing cell death involved in ATF3, RAD21, and caspase3, accompanied by autophagy and apoptosis.
纯度: >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| 规格 | 价格/人民币 | 库存 | 数量 |
| 2MG | ￥575 | 有现货 |
|
| 5MG | ￥956 | 有现货 |
|
| 10MG | ￥1604 | 有现货 |
|
| 25MG | ￥3013 | 有现货 |
|
| 50MG | ￥4504 | 有现货 |
|
| 100MG | ￥6342 | 有现货 |
|
| 500MG | ￥13365 | 有现货 |
|
| 1G | 获取报价 | 有现货 |
|
生物学信息
-
产品名称LYN-1604 dihydrochloride
-
注意事项本公司产品仅用于科研实验，不得用于人体或动物的临床与诊断
-
产品简述LYN-1604 is a novel ULK1 activator.It inducing cell death involved in ATF3, RAD21, and caspase3, accompanied by autophagy and apoptosis.
-
产品描述LYN-1604 is a novel ULK1 activator.It inducing cell death involved in ATF3, RAD21, and caspase3, accompanied by autophagy and apoptosis.(In Vitro):LYN-1604, to be the best candidate for a ULK1 agonist.?LYN-1604 induced cell death involved in ATF3, RAD21, and caspase3, accompanied by autophagy and apoptosis.?LYN-1604 has potential for good therapeutic effects on TNBC by targeting ULK1-modulated cell death in vivo
-
同义词LYN-1604 2HCl（2216753-86-3 free basel);
-
通路Apoptosis
-
靶点Apoptosis
-
受体——
-
研究领域——
-
适应症——
化学信息
-
CAS Number2310109-38-5
-
分子量657.5
-
分子式C33H45Cl4N3O2
-
纯度>98% (HPLC)
-
溶解度——
-
SMILES[H]Cl.[H]Cl.ClC1=CC=C(C(OCC2=CC=C3C=CC=CC3=C2)CN4CCN(C(CN(CC(C)C)CC(C)C)=O)CC4)C(Cl)=C1
-
化学全称——
运输与储存
-
储存条件(-20℃)
-
运输条件With Ice Pack
-
稳定性≥ 2 years
参考文献
1.Liang WL, et al. Exploring the chemodiversity and biological activities of the secondary metabolites from the marine fungus Neosartorya pseudofischeri. Mar Drugs. 2014 Nov 24;12(11):5657-76.
产品手册
关联产品
-
Gomisin N
Gomisin N isolated from Schisandra chinensis produces beneficial sedative and hypnotic bioactivity.
-
Ceranib-2
Ceranib-2 is an inhibitor of ceramidase with an IC50 of 28 μM in SKOV3 cells.
-
Manganese chloride
Manganese chloride inhibits proliferation in a dose- and time-dependent manner induces G0/G1 and S phase arrest and apoptosis in A549 cells.
